Max in WT synaptoneurosomes, suggesting that Src signaling can be downregulated in KI synapses. Then again, our capability to rescue SERT functionality in KI midbrain synaptoneurosomes because of the inhibition of FAK implies elevated FAK signaling downstream in the Pro32Pro33 mutant, as verified by enhanced pFAK localization in five-HT synapses. https://pro3333322.bleepblogs.com/32811285/how-pro33-can-save-you-time-stress-and-money