Max in WT synaptoneurosomes, suggesting that Src signaling can be downregulated in KI synapses. However, our ability to rescue SERT functionality in KI midbrain synaptoneurosomes with the inhibition of FAK signifies elevated FAK signaling downstream with the Pro32Pro33 mutant, as confirmed by amplified pFAK localization in five-HT synapses. Our facts https://shigesatoy097dna8.azuria-wiki.com/user